A 45 years old type II diabetic male had a posterior choroidal rupture secondary to traumatic ocular rupture originated by blunt trauma in the left eye. One year after the trauma the patient developed a choroidal neovascular membrane in the edge of the choroidal rupture. He was treated with intravitreal injections of bevacizumab as needed during two years until the CNVM had no apparent vascular activity and a fibrotic scar is present.

Most of the patients with CNVM secondary to choroidal ruptures present a prompt regression of the lesions with the use of intravitreal anti-VEGF drugs. However this patient needed twelve injections of bevacizumab until the membrane slows its activity. The combination of multiple diseases (diabetes and trauma) may be a risk factor to the poor control of vascular activity of this particular case.